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Fig. 3. (A) Confocal images of laminin staining (green) and human nuclei (HuNu) staining (red) with a DAPI nuclear marker (blue) in ischemic rats with and without hUCB-MSC treatment. Angiogenesis in the ischemic brain. hUCB-MSCs may induce post-ischemic neuroprotection by increasing angiogenesis. HuNu- or laminin-positive cell distribution was dominant in all hUCB-MSC-treated groups (G19, G16, and G5) compared to the control group at day 28 after MCAO. (B) hUCB-MSC treatment increased the number of laminin-positive cells compared to the control group. Scale bar=20 µm.
Exp Neurobiol 2015;24:358~365
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