Fig. 1. Initial brain magnetic resonance imaging (MRI) using fluid-attenuated inversion recovery and diffusion weighted MRI revealed abnormal high signal-intensity lesion lesions with gadolinium enhancement on T1-weighted MRI in the right caudate nucleus which was consistent with subacute cerebral infarction. This cerebral lesions were diagnosed as vasculitic cerebral infarction caused by eosinophilic granulomatosis with polyangiitis (EGPA) (A). After immune therapy with a high-dose steroid and cyclophosphamide, a follow-up brain MRI showed marked improvement compared to the previous MRI (B). Intracranial MR angiography and neck angiography were normal (C).
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