Highlights
  • Short Communication | October 31, 2021

    A deafness gene TMEM43 has been previously shown to interact with connexin channels. In this study, Jang et al. demonstrate TMEM43 interaction with a K2P channel, KCNK3 (TASK-1), in the cochlea.

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    A deafness gene TMEM43 has been previously shown to interact with connexin channels. In this study, Jang et al. demonstrate TMEM43 interaction with a K2P channel, KCNK3 (TASK-1), in the cochlea.
    Minwoo Wendy Jang, Tai Young Kim, Kushal Sharma et al.
  • Original Article | October 31, 2021

    This study presents a possible role of neuronal TLR2 in pulpitis by revealing upregulation of TLR2 expression in a subset of DPA neurons after 24 h following dental pulp injury.

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    This study presents a possible role of neuronal TLR2 in pulpitis by revealing upregulation of TLR2 expression in a subset of DPA neurons after 24 h following dental pulp injury.
    Pa Reum Lee, Jin-Hee Lee, Ji Min Park and Seog Bae Oh
  • Original Article | October 31, 2021

    We found that SIRT3 played a critical role in the cell death induced by oxidative stress by regulating mitochondrial function in mammalian neurons and astrocytes as well as a Drosophila model. Targeting SIRT3 may represent a novel therapeutic intervention for oxidative stress-associated neurodegenerative diseases.

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    We found that SIRT3 played a critical role in the cell death induced by oxidative stress by regulating mitochondrial function in mammalian neurons and astrocytes as well as a Drosophila model. Targeting SIRT3 may represent a novel therapeutic intervention for oxidative stress-associated neurodegenerative diseases.
    Shinrye Lee, Yu-Mi Jeon, Myungjin Jo and Hyung-Jun Kim
  • Original Article | October 31, 2021

    We created precise photothrombotic infarct lesions in the internal capsule of cynomolgus macaques, based on data obtained from electrical mapping of hand motor cortex and viral tracing of the hand motor fibers. Our infarct lesioning technique resulted in persistent motor deficits. A robust capsular infarct model in non-human primates is important for investigating the pathophysiology of subcortical white-matter strokes and potential rehabilitation methods.

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    We created precise photothrombotic infarct lesions in the internal capsule of cynomolgus macaques, based on data obtained from electrical mapping of hand motor cortex and viral tracing of the hand motor fibers. Our infarct lesioning technique resulted in persistent motor deficits. A robust capsular infarct model in non-human primates is important for investigating the pathophysiology of subcortical white-matter strokes and potential rehabilitation methods.
    Hyung-Sun Kim, Jeong Ho Hwang, Su-Cheol Han et al.
  • Original Article | October 31, 2021

    Diffusion measure comparison of basal ganglia and ventral tegmental area structure and diffusion tractography, areas which have historically shown neuronal degeneration in Parkinson’s disease.

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    Diffusion measure comparison of basal ganglia and ventral tegmental area structure and diffusion tractography, areas which have historically shown neuronal degeneration in Parkinson’s disease.
    Jae-Hyuk Shim and Hyeon-Man Baek
Vol.30 No.5 | October 31, 2021

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We created precise photothrombotic infarct lesions in the internal capsule of cynomolgus macaques, based on data obtained from electrical mapping of hand motor cortex and viral tracing of the hand motor fibers. Our infarct lesioning technique resulted in persistent motor deficits. A robust capsular infarct model in non-human primates is important for investigating the pathophysiology of subcortical white-matter strokes and potential rehabilitation methods.